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Furthermore, several properly founded adverse prognostic markers, which include U-CLL, ATM aberrations or NOTCH1/BIRC3 mutations, shed their negative effect in people handled with VO. The sole element that remained predictive of a shorter progression-no cost survival In this particular cohort of patients was TP53 aberrations.112 Finally, the choice BTK inhibitor acalabrutinib was lately authorised through the FDA (not by the EMA nevertheless) as frontline therapy in view of the outcome of the phase III demo evaluating MBL77 acalabrutinib versus ClbO.114
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